non targeting control small guide rna sgrna (Addgene inc)
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Addgene inc
non targeting control small guide rna sgrna
Non Targeting Control Small Guide Rna Sgrna, supplied by Addgene inc, used in various techniques. Bioz Stars score: 94/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/non+targeting+control+small+guide+rna+sgrna/non-targeting+control+gRNA+(BRDN0001145885)+(Plasmid+%2380196)/pm40301689-353-9-21
Average 94 stars, based on 26 article reviews
Non Targeting Control Small Guide Rna Sgrna, supplied by Addgene inc, used in various techniques. Bioz Stars score: 94/100, based on 26 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/non+targeting+control+small+guide+rna+sgrna/non-targeting+control+gRNA+(BRDN0001145885)+(Plasmid+%2380196)/pm40301689-353-9-21
Average 94 stars, based on 26 article reviews
non targeting control small guide rna sgrna - by Bioz Stars,
2026-10
94/100 stars
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Construct:Article Title: ARID5A orchestrates cardiac aging and inflammation through MAVS mRNA stabilization. Article Snippet: Elucidating the regulatory mechanisms of human cardiac aging remains a great challenge.. Here, using human heart tissues from 74 individuals ranging from young (≤35 years) to old (≥65 years), we provide an overview of the histological, cellular and molecular alterations underpinning the aging of human hearts.. We decoded aging-related gene expression changes at single-cell resolution and identified increased inflammation as the key event, driven by upregulation of ARID5A, an RNA-binding protein. Activation Assay:Article Title: ARID5A orchestrates cardiac aging and inflammation through MAVS mRNA stabilization. Article Snippet: Elucidating the regulatory mechanisms of human cardiac aging remains a great challenge.. Here, using human heart tissues from 74 individuals ranging from young (≤35 years) to old (≥65 years), we provide an overview of the histological, cellular and molecular alterations underpinning the aging of human hearts.. We decoded aging-related gene expression changes at single-cell resolution and identified increased inflammation as the key event, driven by upregulation of ARID5A, an RNA-binding protein. Control:Article Title: ARID5A orchestrates cardiac aging and inflammation through MAVS mRNA stabilization. Article Snippet: Elucidating the regulatory mechanisms of human cardiac aging remains a great challenge.. Here, using human heart tissues from 74 individuals ranging from young (≤35 years) to old (≥65 years), we provide an overview of the histological, cellular and molecular alterations underpinning the aging of human hearts.. We decoded aging-related gene expression changes at single-cell resolution and identified increased inflammation as the key event, driven by upregulation of ARID5A, an RNA-binding protein. Clone Assay:Article Title: ARID5A orchestrates cardiac aging and inflammation through MAVS mRNA stabilization. Article Snippet: Elucidating the regulatory mechanisms of human cardiac aging remains a great challenge.. Here, using human heart tissues from 74 individuals ranging from young (≤35 years) to old (≥65 years), we provide an overview of the histological, cellular and molecular alterations underpinning the aging of human hearts.. We decoded aging-related gene expression changes at single-cell resolution and identified increased inflammation as the key event, driven by upregulation of ARID5A, an RNA-binding protein. Plasmid Preparation:Article Title: ARID5A orchestrates cardiac aging and inflammation through MAVS mRNA stabilization. Article Snippet: Elucidating the regulatory mechanisms of human cardiac aging remains a great challenge.. Here, using human heart tissues from 74 individuals ranging from young (≤35 years) to old (≥65 years), we provide an overview of the histological, cellular and molecular alterations underpinning the aging of human hearts.. We decoded aging-related gene expression changes at single-cell resolution and identified increased inflammation as the key event, driven by upregulation of ARID5A, an RNA-binding protein. |